Single leg jumping with it's high impact forces and dependence on the elasticity of muscles and tendons works best for young athletes. With increasing age, the tendons and muscles lose their elasticity and springiness and the risk of injury gets higher and higher. That's why a lot of basketball players start to rely more and more on their two-foot jump as they get older. And the winner of the Olympic high jumping contest are almost always below 30.
I think it’s the sort of thing that a lot of kids probably fantasize about, me included. Just like you might want to become an astronaut or something like that. I was always one of the tallest kids in my class, but I never really tried to dunk. And so as an adult, you start wondering a little bit about what sorts of things you left on the table, that you never really tried your hand at. And I got it into my head that I’d pick up this childhood fantasy of mine and see if I could dunk.
Dunking was banned in the NCAA from 1967 to 1976. Many people have attributed this to the dominance of the then-college phenomenon Lew Alcindor (now known as Kareem Abdul-Jabbar); the no-dunking rule is sometimes referred to as the "Lew Alcindor rule." Many others have also attributed the ban as having racial motivations, as at the time most of the prominent dunkers in college basketball were African-American, and the ban took place less than a year after a Texas Western team with an all-black starting lineup beat an all-white Kentucky team to win the national championship. Under head coach Guy Lewis, Houston (with Elvin Hayes) made considerable use of the "stuff" shot on their way to the Final Four in 1967.
I am 5''11 with a 43 inch vertical I am a freshman and I play on the varsity team as a point gaurd I can do 360''s and now a 540 I want to tell you how I can dunk all I did was watch Vince carter and watch the motion he does and I did the same motion and I never thought I could dunk until the beginning year of 8th grade now I am a freshman posterizing 11 and 12th graders.
Seventy-nine years later, the feat that Daley unwittingly named “the dunk” still flabbergasts. But how it felt to Fortenberry, a pioneering barnstormer whose name we’ve forgotten despite the gold medal he and his teammates won in 1936, remains a mystery. “He never talked about being the first person to dunk and all that,” says 65-year-old Oliver Fortenberry, the only son of Big Joe, who died in ’93. Indeed, the famous dunkers throughout history have been either reticent on the subject or unable to adequately express how it felt to show Dr. Naismith that he’d nailed his peach baskets too low. After more than a year of rigorous research on the subject, I’ve concluded that the inadequacies of modern language—not the ineloquence of the dunk’s practitioners—are at fault. In the eight decades since Fortenberry rocked the rim, words have repeatedly fallen short in describing the only method of scoring, in any sport, that both ignores one of its game’s earliest tenets and, in its very execution, carries a defiant anger.
When performing a vertical jump, the athlete exerts force at the low back, hip, knee, and ankle joints. The spine flexes as the athlete squats downwards, and then is extended by the spinal erectors over the course of the jump. The hip extensors (gluteus maximus, hamstrings, and adductor magnus) work to move the trunk and the thigh apart, which pushes the torso up and backwards. Meanwhile, the knee extensors (quadriceps) contract to extend the knee, and the calf muscles contract to move the shin backwards, towards the vertical.
I mean, I think you can probably improve your vertical some in a month. I think, though, that for most normal people who aren’t teenagers who are trying out for their basketball team, who don’t have all that time on their hands, I think there’s a much saner way to go about it, where you’re steadily improving your vertical over a period of time. You know, there’s a lot of this kind of slightly crazy, kamikaze, self-improvement type of thing, whether it’s trying to jump higher or do anything else. I’m sure those things work to some extent, but it’s not the way I would have wanted to go about it.
A total of 1679 patients were enrolled — 858 in the dopamine group and 821 in the norepinephrine group (Figure 1). All patients were followed to day 28; data on the outcome during the stay in the hospital were available for 1656 patients (98.6%), data on the 6-month outcome for 1443 patients (85.9%), and data on the 12-month outcome for 1036 patients (61.7%). There were no significant differences between the two groups with regard to most of the baseline characteristics (Table 1); there were small differences, which were of questionable clinical relevance, in the heart rate, partial pressure of arterial carbon dioxide (PaCO2), arterial oxygen saturation (SaO2), and ratio of partial pressure of arterial oxygen (PaO2) to fraction of inspired oxygen (FIO2). The type of shock that was seen most frequently was septic shock (in 1044 patients [62.2%]), followed by cardiogenic shock (in 280 patients [16.7%]) and hypovolemic shock (in 263 patients [15.7%]). The sources of sepsis are detailed in Table 2 in the Supplementary Appendix. Hydrocortisone was administered in 344 patients who received dopamine (40.1%) and in 326 patients who received norepinephrine (39.7%). Among patients with septic shock, recombinant activated human protein C was administered in 102 patients in the dopamine group (18.8%) and 96 patients in the norepinephrine group (19.1%).
If anything came to surprise me about this journey, it was the sheer volume of physical pain involved. I had taken on impressive physical feats before. I had run a sub-3:30 marathon back in 2003 (my first and only attempt) after putting in the hundreds of training miles required. I’d done some of the most grueling weight training on offer, most of it either on the beach or at The Yard, a nearby temple of athletic performance where Maria Sharapova, Kobe Bryant and Tom Brady, among many others, have kneeled with exhaustion. But the physical toll of trying to dunk made the marathon and the semipro football and the parenting and everything else I’d ever attempted seem like mere rubber band snaps to the wrist. The lifting didn’t hurt as much as the jumping, the banging of my quadragenarian appendages into the ground, taking off and landing 50 to 200 times a day. My legs never got used to this bludgeoning, never got better at recovering from it, despite my daily foam-rollering, stretching, icing and hydrating. Even on my off days, a quick game of tag with my kids or a bike ride to the park meant daggers in my thighs and a gait like Fred Sanford’s.
I sent a video of my soccer ball dunk to Todd, the #fivefivedunker, who informed me that I was leading with the wrong leg. I’d been taking my last big step with my left foot, which, as a righty, was like swinging a bat cross-handed. A few days later I encountered a blogger and 43-year-old dunker named Andy Nicholson who showed me, among many other things, that I wasn’t the only one with blood on my hands. Nicholson was one of dozens of YouTubers, young and old (mostly young), who were documenting online their attempts to dunk. “Yes!” he yelled over the phone when I told him about the open sores on my fingers. “Those are badges of honor!”
The rate of death at 28 days in this study was close to 50%, which is to be expected in a study with very few exclusion criteria and is similar to the rate in previous observational studies.3,9,21-24 Our trial was a pragmatic study that included all patients who were treated for shock states, and therefore, it has high external validity. The study design allowed for maximal exposure to the study drug, since we included patients who had received open-label vasopressors for a maximum of 4 hours before randomization and since during the 28-day study period, the study drug was withdrawn last when patients were weaned from vasopressor therapies and was resumed first if resumption of vasopressor therapy was necessary.
Use a smaller ball. It's much easier, when you're first starting out, to try dunking with a smaller ball. You'll be able to palm it more easily and control your approach, making the maneuver more satisfying and your practice closer to the real thing. Continue dribbling and shooting exercises with the appropriate-sized ball so you're not getting too used to the "wrong" size, but keep a small ball around for your sick dunks.
In the Noble Asylum's control room, Dr. Hellstrom (a devastating portrayal by Ona Zee) is browsing through the reports of missing Lillian Mangrove (a welcome return for Tyffany Million), the now catatonic Stevens' psychiatrist who went missing right after first examining him. She has been found in a state of severe shock, nursed back to health at the institution and is currently running a psycho-tracking agency, kicking serious nut case butt in attempts to retrieve runaway crazies. Subscribing to the beneficial qualities of shock treatment (hence the title), Hellstrom reactivates Stevens who drags an innocent young nurse tellingly also named Gwen (succulent Shayla LaVeaux) into the dark recesses of his twisted mind, vowing to free her only if the doctors agree to discharge him from their madhouse...
Other obstruction-dunks are worth noting: Haneef Munir performed a Dubble-Up, dunking with his right-hand and then caught and dunked a second ball with his left hand—a yet to be duplicated dunk pioneered by Jordan Kilganon on a lower, non-regulation rim. Jordan Kilganon, a Canadian athlete, approached from the baseline a person standing, holding the ball above their head. Kilganon leaped, controlled the ball in front of his torso and raised it above the horizontal plane of the rim before bringing the ball downward into the hoop and hooking both elbows on and hanging from the rim.
A predefined subgroup analysis was conducted according to the type of shock — septic shock, which occurred in 1044 patients (542 in the dopamine group and 502 in the norepinephrine group); cardiogenic shock, which occurred in 280 patients (135 in the dopamine group and 145 in the norepinephrine group); or hypovolemic shock, which occurred in 263 patients (138 in the dopamine group and 125 in the norepinephrine group). The overall effect of treatment did not differ significantly among these subgroups (P=0.87 for interaction), although the rate of death at 28 days was significantly higher among patients with cardiogenic shock who were treated with dopamine than among those with cardiogenic shock who were treated with norepinephrine (P=0.03) (Figure 3). The Kaplan–Meier curves for the subgroup analysis according to type of shock are shown in Figure 7 in the Supplementary Appendix.
From Jordan to Lebron, even Yao Ming, nothing elicits more awe and applause than a dunk. As one of the highest percentage field goals one can attempt in basketball, this is a move that's worth mastering. While it doesn't hurt to be taller, you can build up both the muscles and skills required to execute this famous feat on the court, regardless of your height and experience. See Step 1 for more information.
The boundary for stopping the trial owing to the lack of evidence of a difference between treatments at a P value of 0.05 was crossed (Figure 5 in the Supplementary Appendix). There were no significant differences between the groups in the rate of death at 28 days or in the rates of death in the ICU, in the hospital, at 6 months, or at 12 months (Table 2). Kaplan–Meier curves for estimated survival showed no significant differences in the outcome (Figure 2). Cox proportional-hazards analyses that included the APACHE II score, sex, and other relevant variables yielded similar results (Figure 6 in the Supplementary Appendix). There were more days without need for the trial drug and more days without need for open-label vasopressors in the norepinephrine group than in the dopamine group, but there were no significant differences between the groups in the number of days without need for ICU care and in the number of days without need for organ support (Table 3). There were no significant differences in the causes of death between the two groups, although death from refractory shock occurred more frequently in the group of patients treated with dopamine than in the group treated with norepinephrine (P=0.05).
Typically, struts consists of a coil spring to support the vehicle's weight, a strut housing to provide rigid structural support for the assembly, and a damping unit within the strut housing to control spring and suspension movement. The bottom of the strut body attaches to the steering knuckle, which in turn connects to a lower control arm through a lower ball joint.
At pickup the next night, buoyed by the previous day’s accomplishment, I found a regulation ball that had good grip, one I could palm, and in between games, when no one was looking, I dunked for the first time in eleven years. If some dunks are described as thunderous, this one could be best described as a gentle fart in the breeze. But a dunk’s a dunk—and I had dunked.
An important component of maximizing height in a vertical jump is attributed to the use of counter-movements of the legs and arm swings prior to take off, as both of these actions have been shown to significantly increase the body’s center of mass rise. The counter-movement of the legs, a quick bend of the knees which lowers the center of mass prior to springing upwards, has been shown to improve jump height by 12% compared to jumping without the counter-movement. This is attributed to the stretch shortening cycle of the leg muscles enabling the muscles to create more contractile energy. Furthermore, jump height can be increased another 10% by executing arm swings during the take off phase of the jump compared to if no arm swings are utilized. This involves lowering the arms distally and posteriorly during the leg counter-movements, and powerfully thrusting the arms up and over the head as the leg extension phase begins. As the arms complete the swinging movement they pull up on the lower body causing the lower musculature to contract more rapidly, hence aiding in greater jump height. Despite these increases due to technical adjustments, it appears as if optimizing both the force producing and elastic properties of the musculotendinous system in the lower limbs is largely determined by genetics and partially mutable through resistance exercise training.