After four months of failing to pull off anything even resembling a real dunk, the planets aligned on Aug. 9: After at least 19 failed attempts that afternoon, I dunked a soccer ball on a middle school court whose rim measured 9' 11". (The original basketball, incidentally, was a soccer ball, property of Dr. Naismith’s employer, Springfield College.) Video from that afternoon shows me standing there, looking confused, in the moment afterward. Did that just happen? Failing had become so routine that even this small success felt foreign.
Unfortunately, I’m not the 6' 7" son of a Hall of Famer, so I had to resort to desperate devices—like Hennessy, an infamous and inexpensive cognac that, according to one of the two NBA players who recommended it to me, “will give you that Yah! That bounce. That little bit of meanness you need.” The little minibar-sized bottle that I downed 30 minutes into an intense session of dunk attempts on a sweltering day last summer, had no effect other than scorching my esophagus, giving me a headache and releasing from my pores an aura that, as my six-year-old put it that evening, “smells like medicine.”
A predefined subgroup analysis was conducted according to the type of shock — septic shock, which occurred in 1044 patients (542 in the dopamine group and 502 in the norepinephrine group); cardiogenic shock, which occurred in 280 patients (135 in the dopamine group and 145 in the norepinephrine group); or hypovolemic shock, which occurred in 263 patients (138 in the dopamine group and 125 in the norepinephrine group). The overall effect of treatment did not differ significantly among these subgroups (P=0.87 for interaction), although the rate of death at 28 days was significantly higher among patients with cardiogenic shock who were treated with dopamine than among those with cardiogenic shock who were treated with norepinephrine (P=0.03) (Figure 3). The Kaplan–Meier curves for the subgroup analysis according to type of shock are shown in Figure 7 in the Supplementary Appendix.
A total of 1679 patients were enrolled — 858 in the dopamine group and 821 in the norepinephrine group (Figure 1). All patients were followed to day 28; data on the outcome during the stay in the hospital were available for 1656 patients (98.6%), data on the 6-month outcome for 1443 patients (85.9%), and data on the 12-month outcome for 1036 patients (61.7%). There were no significant differences between the two groups with regard to most of the baseline characteristics (Table 1); there were small differences, which were of questionable clinical relevance, in the heart rate, partial pressure of arterial carbon dioxide (PaCO2), arterial oxygen saturation (SaO2), and ratio of partial pressure of arterial oxygen (PaO2) to fraction of inspired oxygen (FIO2). The type of shock that was seen most frequently was septic shock (in 1044 patients [62.2%]), followed by cardiogenic shock (in 280 patients [16.7%]) and hypovolemic shock (in 263 patients [15.7%]). The sources of sepsis are detailed in Table 2 in the Supplementary Appendix. Hydrocortisone was administered in 344 patients who received dopamine (40.1%) and in 326 patients who received norepinephrine (39.7%). Among patients with septic shock, recombinant activated human protein C was administered in 102 patients in the dopamine group (18.8%) and 96 patients in the norepinephrine group (19.1%).

All data were analyzed according to the intention-to-treat principle. Differences in the primary outcome were analyzed with the use of an unadjusted chi-square test. Results are presented as absolute and relative risks and 95% confidence intervals. Kaplan–Meier curves for estimated survival were compared with the use of a log-rank test. A Cox proportional-hazards regression model was used to evaluate the influence of potential confounding factors on the outcome (factors were selected if the P value in the univariate analysis was <0.20).
Turn on the windmill. As you approach, bring the ball into your abdomen and back, extending your arm behind your body and up in a circular fashion, like a windmill spinning. At the apex of your jump, bring your arm all the way around to throw it down like a boss. Dominique Wilkins, the Dunkmaster General of the 90s, used to blow crowds away with this spectacular dunk.
I went through this progression, too. I went from touching the middle of the net at 12 years old, to dunking a basketball at 14 years old, to doing serious acrobatic 360-degree dunks at 17 years old. In college, my personal record for the vertical leap was 40 inches. At my peak, I was able to touch the top of the square on a regulation backboard, about 11.5 feet from the ground. Even now, in my thirties, I can dunk a basketball while standing underneath the basket—no run up required. I owe it all to the power of the vertical jump.
Jumping Rope – A skipping rope is the only piece of equipment involved in the program. If you don’t have one a piece of rope will do just fine. If you don’t have a piece of rope either jumping up and down on the spot without much bending in the knees will achieve a similar result. Jumping rope involves holding a rope with both hands and swinging it around your body continuously.